Researcher Dr Steven Lipshultz, from the Childrens Hospital of Michigan, said that children with dilated cardiomyopathy in their families are generally diagnosed at a younger age than children with dilated cardiomyopathy of an unknown cause (idiopathic). The multi-centre study, which may influence how children are treated around the world, has just been published in Circulation Heart Failure, the journal produced by the American Heart Association. Borrs;., Laboratory Investigation, 2018. More Watchlist Dolichol kinase deficiency (dolkcdg Two new cases and expansion of phenotype 17-Aug-2017 Eric. Study Materials section for this and other study documents. Clinical and genetic issues in familial dilated cardiomyopathy. Kuhl U, Pauschinger M, Seeberg B.
Clinical and mutational spectrum in a cohort of 105 unrelated patients with dilated cardiomyopathy. Gadolinium and nephrogenic systemic fibrosis. Luanne Thomas-Ewald, the CEO of the Childrens Hospital of Michigan, said: With this research we are not only changing how we most appropriately treat children with heart diseases, but we are also changing the way the world thinks about this important issue.
Soluble ST2 reflects hemodynamic stress in non-ischemic heart failure. Use and interpretation youth justice essays of genetic tests in cardiovascular genetics. Broch K, Andreassen AK, Ueland T. Dilated cardiomyopathy is the most common type of cardiomyopathy in infancy, childhood and adolescence. Sakamoto; Emily Moreno; Susan. Single-gene mutations and increased left ventricular wall thickness in the community: the Framingham Heart Study.
Dilated cardiomyopathy research paper